 Stefanie Makowski
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| Title(s) | Postdoctoral Scholar, Medicine |
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| School | Vc-health Sciences-schools |
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| Address | 9500 Gilman Drive #
La Jolla CA 92093
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| vCard | Download vCard |
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 Bibliographic
Altmetrics Details
PMC Citations indicate the number of times the publication was cited by articles in PubMed Central, and the Altmetric score represents citations in news articles and social media.
(Note that publications are often cited in additional ways that are not shown here.)
Fields are based on how the National Library of Medicine (NLM) classifies the publication's journal and might not represent the specific topic of the publication.
Translation tags are based on the publication type and the MeSH terms NLM assigns to the publication.
Some publications (especially newer ones and publications not in PubMed) might not yet be assigned Field or Translation tags.)
Click a Field or Translation tag to filter the publications.
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Efficient Golgi Forward Trafficking Requires GOLPH3-Driven, PI4P-Dependent Membrane Curvature. Dev Cell. 2019 09 09; 50(5):573-585.e5.
Rahajeng J, Kuna RS, Makowski SL, Tran TTT, Buschman MD, Li S, Cheng N, Ng MM, Field SJ. PMID: 31231041.
View in: PubMed Mentions: 2 Fields: Translation: HumansCells
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Neolymphostin A Is a Covalent Phosphoinositide 3-Kinase (PI3K)/Mammalian Target of Rapamycin (mTOR) Dual Inhibitor That Employs an Unusual Electrophilic Vinylogous Ester. J Med Chem. 2018 12 13; 61(23):10463-10472.
Castro-Falcón G, Seiler GS, Demir Ö, Rathinaswamy MK, Hamelin D, Hoffmann RM, Makowski SL, Letzel AC, Field SJ, Burke JE, Amaro RE, Hughes CC. PMID: 30380865.
View in: PubMed Mentions: Fields: Translation: Cells
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Emerging themes of regulation at the Golgi. Curr Opin Cell Biol. 2017 04; 45:17-23.
Makowski SL, Tran TT, Field SJ. PMID: 28213314.
View in: PubMed Mentions: 8 Fields: Translation: HumansAnimalsCells
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NK Cell Maturation and Cytotoxicity Are Controlled by the Intramembrane Aspartyl Protease SPPL3. J Immunol. 2016 Mar 15; 196(6):2614-26.
Hamblet CE, Makowski SL, Tritapoe JM, Pomerantz JL. PMID: 26851218.
View in: PubMed Mentions: 2 Fields: Translation: AnimalsCells
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A protease-independent function for SPPL3 in NFAT activation. Mol Cell Biol. 2015 Jan; 35(2):451-67.
Makowski SL, Wang Z, Pomerantz JL. PMID: 25384971.
View in: PubMed Mentions: 6 Fields: Translation: HumansCells
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Oncogenic CARD11 mutations induce hyperactive signaling by disrupting autoinhibition by the PKC-responsive inhibitory domain. Biochemistry. 2010 Sep 28; 49(38):8240-50.
Lamason RL, McCully RR, Lew SM, Pomerantz JL. PMID: 20799731.
View in: PubMed Mentions: 38 Fields: Translation: HumansCells
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Transcriptional target-based expression cloning of immunoregulatory molecules. Immunol Res. 2010 Jul; 47(1-3):172-8.
Lamason RL, Lew SM, Pomerantz JL. PMID: 20069388.
View in: PubMed Mentions: 2 Fields: Translation: HumansCells
This graph shows the total number of publications by year. To see the data as text, click here.
This graph shows the total number of publications by year. To return to the graph, click here.
| Year | Publications |
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| 2010 | 2 | | 2014 | 1 | | 2016 | 1 | | 2017 | 1 | | 2018 | 1 | | 2019 | 1 |
This graph shows the number and percent of publications by field.
Fields are based on how the National Library of Medicine (NLM) classifies the publications' journals and might not represent the specific topics of the publications.
Note that an individual publication can be assigned to more than one field. As a result, the publication counts in this graph might add up to more than the number of publications the person has written.
To see the data as text, click here.
This graph shows the number and percent of publications by field.
Fields are based on how the National Library of Medicine (NLM) classifies the publications' journals and might not represent the specific topics of the publications.
Note that an individual publication can be assigned to more than one field. As a result, the publication counts in this graph might add up to more than the number of publications the person has written.
To see the data as text, click here.
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